Curriculum vitae - Jan-Wilhelm Kornfeld

Education and Professional Experience

• 1997-2002
  Undergraduate Studies of Biochemistry), University of Hannover, Hannover, Germany
• 2002-2003
  University degree in Biochemistry, Faculty for Natural Sciences, Medical School Hannover (MHH), Hannover, Germany
• 2002-2003
  Full-Time Research Associate, Department of Tumor Biology, University Hospital Hamburg-Eppendorf, Hamburg, Germany
• 2006-2008
  PhD Student, Ludwig-Boltzmann-Institute for Cancer Research (LBICR), Vienna, Austria
• 2008
  PhD Dissertation, Faculty for Natural Sciences and Mathematics, University of Kiel, Kiel, Germany
• 2008-2009
  Postdoctoral Fellow, Lab of Prof. Richard Moriggl, LBICR, Vienna, Austria
• 2009-2013
  Postdoctoral Fellow, Lab of Prof. Jens C. Brüning, Institute for Genetics, University of Cologne, Cologne, Germany
• since 2013
  Principal Investigator, Cluster of Excellence: Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany
• since 2014
  Independent Group Leader, Max Planck Institute for Metabolism Research (formerly the MPI for Neurological Research), Cologne, Germany

Professional Memberships and Activities

• since 2008
  Member of the Austrian Association of Molecular Life Sciences and Biotechnology (ÖGMBT)   
• since 2008
  Member of the American Association for Cancer Research (AACR) 
• since 2008
  Supervisor of over 10 PhD, MSc, and BSc students
• 2009-2012
  Member of the CECAD Postdoctoral Steering Committee                 
• since 2013
  Ad-hoc Reviewer for the Journal of Clinical Investigation, Nat Cell Biol, J Clin Invest and Cell Cycle
• since 2013
• Associate Faculty Member, International Graduate School for Development, Health and Diseases (IGS-DHD), Faculty of Biology, University of Cologne, Cologne, Germany
• since 2013
  Associate Faculty Member, Cologne Graduate School for Ageing Research, Cologne, Germany
• 2014
  Member of the CECAD Postdoctoral Steering Committee
• 2014
  Conference Organizer ‘Noncoding RNAs – Biology and Disease’, Cologne, Germany
• 2014
  Ad-hoc Grant Reviewer for the German Research Society (DFG)

Prizes, Awards, and Honors

• 2010-2012
  EMBO Long-term Fellowship, European Molecular Biology Organisation
• 2011-2012
  CECAD Senior Postdoctoral Grant
• since 2014
  Emmy-Noether Awardee, German Research Society (DFG)

Selected Publications

Kornfeld JW, Baitzel B, Könner AC, Nicholls HT, Vogt MC, Herrmanns C, Scheja L, Haumaitre C, Wolf AM, Knippschild U, Seibler J, Cereghini S,
Heeren J, Stoffel M and Brüning JC (2013).Obesity-Induced Overexpression of miR-802 Impairs Glucose Metabolism Via Silencing of Hnf1b.
Nature 7;494(7435):111-5. doi: 10.1038/nature11793.

Kornfeld JW, Isaacs A, Vitart V, Pospisilik JA, Meitinger T, Gyllensten U, Wilson JF, Rudan I, Campbell H, Penninger JM, Sexl V, Moriggl R, van
Duijn C, Pramstaller PP and Hicks AA (2011). Variants in STAT5B associate with serum TC and LDL-C levels. J Clin Endocrinol Metab 96(9):E1496-501.doi: 10.1210/jc.2011-0322

*Mueller KM, *Kornfeld JW, Friedbichler K, Blaas L, Egger G, Esterbauer H, Hasselblatt P, Schlederer M, Haindl S, Wagner KU, Engblom D,
Haemmerle G, Kratky D, Sexl V, Kenner L, Kozlov AV, Terracciano L, Zechner R, Schuetz G, Casanova E, Pospisilik JA, Heim MH and Moriggl R (2011).
Impairment of hepatic growth hormone and glucocorticoid receptor signaling causes steatosis and hepatocellular carcinoma in mice.Hepatology, 54(4):1398-409. doi: 10.1002/hep.24509

*=equal contribution

*Blaas L, *Kornfeld JW, Schramek D, Musteanu M, Zollner G, Gumhold J, Schneller D, Esterbauer H, Mair M, Mikulits W, Eferl R, Moriggl R, Penninger JM, Trauner M and Casanova E (2010). Disruption of the GH-STAT5-IGF-1 axis severely aggravates liver fibrosis in a mouse model of cholestasis.Hepatology, *=equal contribution. 51(4):1319-26. doi: 10.1002/hep.23469

*=equal contribution

*Engblom D, *Kornfeld JW, Schwake L, Tronche F, Reimann A, Beug H, Hennighausen L, Moriggl R and Schütz G (2007). Direct glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression. Genes Dev, 15;21(10):1157-62

*=equal contribution.