Dr. Jan-Wilhelm Kornfeld

Curriculum vitae - Jan-Wilhelm Kornfeld

Education and Professional Experience

  • 1997-2002
    Undergraduate Studies of Biochemistry), University of Hannover, Hannover, Germany
  • 2002-2003
    University degree in Biochemistry, Faculty for Natural Sciences, Medical School Hannover (MHH), Hannover, Germany
  • 2002-2003
    Full-Time Research Associate, Department of Tumor Biology, University Hospital Hamburg-Eppendorf, Hamburg, Germany
  • 2006-2008
    PhD Student, Ludwig-Boltzmann-Institute for Cancer Research (LBICR), Vienna, Austria
  • 2008
    PhD Dissertation, Faculty for Natural Sciences and Mathematics, University of Kiel, Kiel, Germany
  • 2008-2009
    Postdoctoral Fellow, Lab of Prof. Richard Moriggl, LBICR, Vienna, Austria
  • 2009-2013
    Postdoctoral Fellow, Lab of Prof. Jens C. Brüning, Institute for Genetics, University of Cologne, Cologne, Germany
  • since 2013
    Principal Investigator, Cluster of Excellence: Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany
  • since 2014
    Independent Group Leader, Max Planck Institute for Metabolism Research (formerly the MPI for Neurological Research), Cologne, Germany

Professional Memberships and Activities

  • since 2008
    Member of the Austrian Association of Molecular Life Sciences and Biotechnology (ÖGMBT)   
  • since 2008
    Member of the American Association for Cancer Research (AACR) 
  • since 2008
    Supervisor of over 10 PhD, MSc, and BSc students
  • 2009-2012
    Member of the CECAD Postdoctoral Steering Committee                 
  • since 2013
    Ad-hoc Reviewer for the Journal of Clinical Investigation, Nat Cell Biol, J Clin Invest and Cell Cycle
  • since 2013
  • Associate Faculty Member, International Graduate School for Development, Health and Diseases (IGS-DHD), Faculty of Biology, University of Cologne, Cologne, Germany
  • since 2013
    Associate Faculty Member, Cologne Graduate School for Ageing Research, Cologne, Germany
  • 2014
    Member of the CECAD Postdoctoral Steering Committee
  • 2014
    Conference Organizer ‘Noncoding RNAs – Biology and Disease’, Cologne, Germany
  • 2014
    Ad-hoc Grant Reviewer for the German Research Society (DFG)

Prizes, Awards, and Honors

Selected Publications

Kornfeld JW, Baitzel B, Könner AC, Nicholls HT, Vogt MC, Herrmanns C, Scheja L, Haumaitre C, Wolf AM, Knippschild U, Seibler J, Cereghini S,
Heeren J, Stoffel M and Brüning JC (2013).Obesity-Induced Overexpression of miR-802 Impairs Glucose Metabolism Via Silencing of Hnf1b.
Nature 7;494(7435):111-5. doi: 10.1038/nature11793.

Kornfeld JW, Isaacs A, Vitart V, Pospisilik JA, Meitinger T, Gyllensten U, Wilson JF, Rudan I, Campbell H, Penninger JM, Sexl V, Moriggl R, van
Duijn C, Pramstaller PP and Hicks AA (2011). Variants in STAT5B associate with serum TC and LDL-C levels. J Clin Endocrinol Metab 96(9):E1496-501.doi: 10.1210/jc.2011-0322

*Mueller KM, *Kornfeld JW, Friedbichler K, Blaas L, Egger G, Esterbauer H, Hasselblatt P, Schlederer M, Haindl S, Wagner KU, Engblom D,
Haemmerle G, Kratky D, Sexl V, Kenner L, Kozlov AV, Terracciano L, Zechner R, Schuetz G, Casanova E, Pospisilik JA, Heim MH and Moriggl R (2011).
Impairment of hepatic growth hormone and glucocorticoid receptor signaling causes steatosis and hepatocellular carcinoma in mice.Hepatology, 54(4):1398-409. doi: 10.1002/hep.24509

*=equal contribution

*Blaas L, *Kornfeld JW, Schramek D, Musteanu M, Zollner G, Gumhold J, Schneller D, Esterbauer H, Mair M, Mikulits W, Eferl R, Moriggl R, Penninger JM, Trauner M and Casanova E (2010). Disruption of the GH-STAT5-IGF-1 axis severely aggravates liver fibrosis in a mouse model of cholestasis.Hepatology, *=equal contribution. 51(4):1319-26. doi: 10.1002/hep.23469

*=equal contribution

*Engblom D, *Kornfeld JW, Schwake L, Tronche F, Reimann A, Beug H, Hennighausen L, Moriggl R and Schütz G (2007). Direct glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression. Genes Dev, 15;21(10):1157-62

*=equal contribution.

 
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